FMS Like tyrosine Kinase 3 (FLT3) gene mutations in Acute Myeloid Leukemia (AML) In Patients with Low or Intermediate risk Karyotypes
Essam Hamed Elnoshokaty;
Abstract
Both ITD and D835 mutations of the fms-like tyrosine kinase (FLT3) gene cause constitutive activation of the receptor, in the absence of ligand. We have examined a cohort of 60 newly diagnosed AML patients to determine the prevalence of these mutations an
Other data
| Title | FMS Like tyrosine Kinase 3 (FLT3) gene mutations in Acute Myeloid Leukemia (AML) In Patients with Low or Intermediate risk Karyotypes | Other Titles | الطفرات الوراثية فى جين إف إم إس شبيه التيروزين كيناز 3(إف إل ت 3 )فى سرطان الدم الميلودى الحاد فى المرضى ذوى التركيب الكروموزومى الجيد أو المتوسط الخطورة | Authors | Essam Hamed Elnoshokaty | Keywords | FMS Like tyrosine Kinase 3 (FLT3) gene mutations in Acute Myeloid Leukemia (AML) In Patients with Low or Intermediate risk Karyotypes | Issue Date | 2009 | Description | Both ITD and D835 mutations of the fms-like tyrosine kinase (FLT3) gene cause constitutive activation of the receptor, in the absence of ligand. We have examined a cohort of 60 newly diagnosed AML patients to determine the prevalence of these mutations an |
Attached Files
| File | Size | Format | |
|---|---|---|---|
| 91089Untitled.pdf | 12.5 MB | Adobe PDF | View/Open |
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